A hidden clue may explain why some mutated cells become cancerous and others don’t: how fast they divide. A new study from researchers at Sinai Health in Toronto reveals that the total time it takes ...

Johns Hopkins Medicine scientists say they have found a pattern of so-called epigenetic "marks" in a transition state between normal and pancreatic cancer cells in mice, and that the normal cells may ...

New research has shown how brain cells acquire a cancerous mutation and spread across the cortex, opening a new path for ...

The cancer gene MYC camouflages tumours by suppressing alarm signals that normally activate the immune system. This finding ...

Scientists from A*STAR Institute of Molecular and Cell Biology (A*STAR IMCB) have identified why certain lung cancer cells become highly resistant to treatment after developing mutations in a key gene ...

Scientists say they have found a pattern of so-called epigenetic 'marks' in a transition state between normal and pancreatic cancer cells in mice, and that the normal cells may keep at least a ...

Scientists have recently been learning more about the importance of small bits of circular genetic material known as extrachromosomal DNA (ecDNA). These little circles of DNA can hitch a ride with ...

BRCA1 and BRCA2 genes play a crucial role in repairing damaged DNA. Mutations in BRCA1 and BRCA2 substantially increase the likelihood of breast and ovarian cancer, respectively. This new research ...

The research, published today in Nature Communications, focuses on how cancer cells keep their chromosomes intact so they can continue dividing relentlessly. Normally, every time a cell divides, the ...

Some cancer cells don't die; they go quiet, like seeds lying dormant in the soil. These "sleeper cells," scattered throughout the body, can stay inactive for years. But when the body faces a ...